<oai_dc:dc xmlns:dc="http://purl.org/dc/elements/1.1/" xmlns:oai_dc="http://www.openarchives.org/OAI/2.0/oai_dc/" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xsi:schemaLocation="http://www.openarchives.org/OAI/2.0/oai_dc/ http://www.openarchives.org/OAI/2.0/oai_dc.xsd">
  <dc:creator>Müller V</dc:creator>
  <dc:creator>Vigueras-Gómez JF</dc:creator>
  <dc:creator>Bonhoeffer S</dc:creator>
  <dc:date>2002</dc:date>
  <dc:description xmlns:ns0="xml" ns0:lang="en">Antiviral therapy induces a rapid drop in human immunodeficiency virus type 1 viremia, but the decline of virus levels decelerates over time. Mathematical modeling demonstrates that the source of residual virus production might be a single compartment of latently infected cells with an extended distribution of activation rates.</dc:description>
  <dc:format>application/pdf</dc:format>
  <dc:identifier>https://sonar.ch/global/documents/139899</dc:identifier>
  <dc:language>eng</dc:language>
  <dc:relation>info:eu-repo/semantics/altIdentifier/doi/10.1128/jvi.76.17.8963-8965.2002</dc:relation>
  <dc:relation>info:eu-repo/semantics/altIdentifier/pmid/12163616</dc:relation>
  <dc:rights>info:eu-repo/semantics/openAccess</dc:rights>
  <dc:source>Journal of virology. - 2002</dc:source>
  <dc:subject xmlns:ns1="xml" ns1:lang="en">Antiretroviral Therapy, Highly Active</dc:subject>
  <dc:subject xmlns:ns2="xml" ns2:lang="en">CD4-Positive T-Lymphocytes</dc:subject>
  <dc:subject xmlns:ns3="xml" ns3:lang="en">HIV Infections</dc:subject>
  <dc:subject xmlns:ns4="xml" ns4:lang="en">HIV-1</dc:subject>
  <dc:subject xmlns:ns5="xml" ns5:lang="en">Humans</dc:subject>
  <dc:subject xmlns:ns6="xml" ns6:lang="en">Models, Biological</dc:subject>
  <dc:subject xmlns:ns7="xml" ns7:lang="en">Time Factors</dc:subject>
  <dc:subject xmlns:ns8="xml" ns8:lang="en">Virus Activation</dc:subject>
  <dc:subject xmlns:ns9="xml" ns9:lang="en">Virus Latency</dc:subject>
  <dc:subject xmlns:ns10="xml" ns10:lang="en">Virus Replication</dc:subject>
  <dc:title xmlns:ns11="xml" ns11:lang="en">Decelerating decay of latently infected cells during prolonged therapy for human immunodeficiency virus type 1 infection.</dc:title>
  <dc:type>http://purl.org/coar/resource_type/c_6501</dc:type>
</oai_dc:dc>