Journal article

Periodontal health and gingival diseases and conditions on an intact and a reduced periodontium: Consensus report of workgroup 1 of the 2017 World Workshop on the Classification of Periodontal and Peri-Implant Diseases and Conditions.

  • Chapple ILC Periodontal Research Group, Institute of Clinical Sciences, College of Medical & Dental Sciences, University of Birmingham, UK.
  • Mealey BL University of Texas Health Science Center at San Antonio, USA.
  • Van Dyke TE The Forsyth Institute, Cambridge, MA, USA.
  • Bartold PM School of Dentistry, University of Adelaide, Australia.
  • Dommisch H Department of Periodontology and Synoptic Dentistry, Charité - Universitätsmedizin Berlin, Germany.
  • Eickholz P Department of Periodontology, Center for Oral Medicine, Johann Wolfgang Goethe-University Frankfurt, Germany.
  • Geisinger ML Department of Periodontology, University of Alabama at Birmingham, USA.
  • Genco RJ Department of Oral Biology, SUNY at Buffalo, NY, USA.
  • Glogauer M Faculty of Dentistry, University of Toronto, Canada.
  • Goldstein M Department of Periodontology, Faculty of Dental Medicine, Hebrew University-Hadassah Medical Center, Jerusalem, Israel.
  • Griffin TJ Periodontal Department, Tufts University School of Dental Medicine, Boston, MA, USA.
  • Holmstrup P Periodontology, Section 1, Faculty of Health and Medical Sciences, University of Copenhagen, Denmark.
  • Johnson GK Department of Periodontology, University of Iowa College of Dentistry, Iowa City, IA, USA.
  • Kapila Y Orofacial Sciences, University of California San Francisco, USA.
  • Lang NP Department of Periodontology, University of Bern, Switzerland.
  • Meyle J Department of Periodontology, University of Giessen, Germany.
  • Murakami S Department of Periodontology, Graduate School of Dentistry, Osaka University, Japan.
  • Plemons J Department of Periodontics, Texas A&M College of Dentistry, Dallas, TX, USA.
  • Romito GA Division of Periodontology, Department of Stomatology, Dental School, University of São Paulo, Brazil.
  • Shapira L Department of Periodontology, Faculty of Dental Medicine, Hebrew University-Hadassah Medical Center, Jerusalem, Israel.
  • Tatakis DN Division of Periodontology, College of Dentistry, Ohio State University, Columbus, OH, USA.
  • Teughels W Department of Oral Health Sciences, Periodontology, KU Leuven & Dentistry, University Hospitals Leuven, Belgium.
  • Trombelli L Research Center for the Study of Periodontal and Peri-Implant Diseases, University of Ferrara, Italy.
  • Walter C Department of Periodontology, Endodontology & Cariology, University Centre for Dental Medicine, University of Basel School of Dentistry, Switzerland.
  • Wimmer G Department of Prosthodontics, School of Dentistry, Medical University Graz, Austria.
  • Xenoudi P Orofacial Sciences, School of Dentistry, University of California San Francisco, USA.
  • Yoshie H Division of Periodontology, Niigata University Graduate School of Medical and Dental Sciences, Japan.
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  • 2018-06-22
Published in:
  • Journal of clinical periodontology. - 2018
Crohn's disease
Hodgkin lymphoma
Melkersson-Rosenthal
Mycobacterium tuberculosis
Neisseria gonorrhoeae
Treponema pallidum
allergic reaction
amalgam tattoo
aspergillosis
biofilm
blastomycosis
calcifying fibroblastic granuloma
candidosis
chemical trauma
clinical health
coccidioidomycosis
condylomata acuminatum
contact allergy
coxsackie virus
dental plaque-induced gingivitis
disease control
disease remission
disease stability
drug-induced gingival enlargement
drug-induced pigmentation
dysbiosis
erythema multiforme
erythroplakia
factitious injury
fibrous epulis
focal epithelial hyperplasia
frictional keratosis
geotricosis
gingival pigmentation
hand foot and mouth
hereditary gingival fibromatosis
herpangina
herpes simplex
histoplasmosis
hyperglycemia
hyposalivation
intact periodontium
leukemia
leukoplakia
lichen planus
local risk factors
lupus erythematosus
melanoplakia
menstrual cycle
modifying factors
molluscum contagiosum
mucormycosis
necrotizing periodontal diseases
non-Hodgkin lymphoma
non-dental plaque-induced gingival conditions
oral contraceptive
orofacial granulomatosis
paracoccidioidomycosis
pemphigoid
pemphigus vulgaris
periodontal disease
peripheral giant cell granuloma
plasma cell gingivitis
predisposing factors
pregnancy
puberty
pyogenic granuloma
reduced periodontium
resolution of inflammation
restoration margins
sarcoidosis
scurvy
smoker's melanosis
smoking
squamous cell carcinoma
squamous cell papilloma
stable periodontitis
streptoccocal gingivitis
symbiosis
systemic risk factors
thermal trauma
toothbrush trauma
varicella zoster
vascular epulis
verruca vulgaris
Consensus
Dental Plaque
Gingivitis
Humans
Periodontitis
Periodontium
English Periodontal health is defined by absence of clinically detectable inflammation. There is a biological level of immune surveillance that is consistent with clinical gingival health and homeostasis. Clinical gingival health may be found in a periodontium that is intact, i.e. without clinical attachment loss or bone loss, and on a reduced periodontium in either a non-periodontitis patient (e.g. in patients with some form of gingival recession or following crown lengthening surgery) or in a patient with a history of periodontitis who is currently periodontally stable. Clinical gingival health can be restored following treatment of gingivitis and periodontitis. However, the treated and stable periodontitis patient with current gingival health remains at increased risk of recurrent periodontitis, and accordingly, must be closely monitored. Two broad categories of gingival diseases include non-dental plaque biofilm-induced gingival diseases and dental plaque-induced gingivitis. Non-dental plaque biofilm-induced gingival diseases include a variety of conditions that are not caused by plaque and usually do not resolve following plaque removal. Such lesions may be manifestations of a systemic condition or may be localized to the oral cavity. Dental plaque-induced gingivitis has a variety of clinical signs and symptoms, and both local predisposing factors and systemic modifying factors can affect its extent, severity, and progression. Dental plaque-induced gingivitis may arise on an intact periodontium or on a reduced periodontium in either a non-periodontitis patient or in a currently stable "periodontitis patient" i.e. successfully treated, in whom clinical inflammation has been eliminated (or substantially reduced). A periodontitis patient with gingival inflammation remains a periodontitis patient (Figure 1), and comprehensive risk assessment and management are imperative to ensure early prevention and/or treatment of recurrent/progressive periodontitis. Precision dental medicine defines a patient-centered approach to care, and therefore, creates differences in the way in which a "case" of gingival health or gingivitis is defined for clinical practice as opposed to epidemiologically in population prevalence surveys. Thus, case definitions of gingival health and gingivitis are presented for both purposes. While gingival health and gingivitis have many clinical features, case definitions are primarily predicated on presence or absence of bleeding on probing. Here we classify gingival health and gingival diseases/conditions, along with a summary table of diagnostic features for defining health and gingivitis in various clinical situations.
Language
  • English
Open access status
bronze
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https://sonar.ch/global/documents/222
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